If it were PGD2.....
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I asked this question a month ago when all this prostaglandin stuff exploded and I never got an answer. It doesn't make sense that transplanted hairs would remain if the frontal area is susceptible to increased levels of PGD2 unless the follicles themselves are resistant. I have never understood why transplanted hairs do not fall out, other than that they are somehow "DHT resistant." If heightened levels of PGD2 exist in my frontal area and that alone causes my baldness then why the hell would transplanted hairs stick around in the same environment?? So frustratingly complex... -
Yes they did test the same scalp from what I recall. The balding areas had elevated PGD2, the non-balding areas didn't.
The most likely answer IMO is that PG imbalance is localised to the affected follicles, and the transplanted hair follicle doesn't have an androgen response so that doesn't start the cascading chain of events that ends in PG imbalance and miniaturised follicles.
But I'm just speculating.Leave a comment:
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Yes they did test the same scalp from what I recall. The balding areas had elevated PGD2, the non-balding areas didn't.
The most likely answer IMO is that PG imbalance is localised to the affected follicles, and the transplanted hair follicle doesn't have an androgen response so that doesn't start the cascading chain of events that ends in PG imbalance and miniaturised follicles.
But I'm just speculating.Leave a comment:
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Leave a comment: