Masturbation can cause more hair loss/DHT to those who are genetically prone to it.

[burtandernie]

I still think no one really knows. What about men that are susceptible to MPB that are athletes that do not suffer insulin resistance and have normal SHBG yet suffer early onset of MPB? I can assure you plenty of men fitting those circumstances exist and kind of contradicts this theory. If this statement were pure fact everyone with MPB would basically be pre-diabetic then, and thats simply not true.
So maybe its evidence towards environment has influence, but its a question of how much? shbg isnt even a constant it could high one month and lower the next from different variables like excercise/drinking alcohol which supposedly raise it

Study A - It's been observed that men with early onset of baldness suffers from insulin resistance (pre-diabetics) and have low level of SHBG

[failly]

Bumping this for new users!

[NeedHairASAP]

Correlation of Carotid Intima Media Thickness and Aortic Stiffness
Index With Androgenetic Alopecia

Aim: Androgenetic alopecia (AGA) is a common health problem which is well associated with
hair loss in both male and female subjects by the effect of androgens under the presence of
genetic predisposition. Recent studies showed that there is an increased risk for coronary artery
disease in AGA patients. The aortic stiffness index (ASI) and the carotid intima media
thickness (CIMT) are accepted as a strong marker for the presence of atherosclerosis. The aim
of this study is to determine the possible correlation of CIMT and ASI with presence of AGA.
Materials and methods: A total of 159 male asymptomatic AGA patients between 18-55 years
old without any history of chronic disease enrolled to the study. Patients were classified in the
means of their AGA stage as group I (stage 1-2) (n=49), group II (stage 3-5) (n=71) and group III
(stage 6) (n=39) according to the Hamilton scale. Echocardiography was used to determine the
elastic properties of aorta assessed by ASI and aortic distensibility (AD) parameters. CIMT was
measured by color Doppler ultrasonography. ASI, AD and mean CIMT were calculated. The
data including the patient’s metabolic profile and anthropometric measurements were recorded.

Results: There was no significant difference between the groups in the means of age, height,
body mass index, and left ventricle ejection fraction. The values of mean ASI, CIMT and systolic
blood pressures were significantly higher in group III than group I and group II statistically
(p<0.001 for all three parameters). On the other hand, AD was significantly higher in group I
compared with group II and III statistically (p<0.001).
Conclusions: ASI and AD are parameters reflecting the aortic elastic properties. In case of an
aortic elasticity deterioration, ASI value increases while AD value decrease. This study concluded
that the patients at higher stages of AGA had higher CIMT and more deteriorated aortic
elasticity which are indicators of atherosclerosis. Thus, we assumed that patients with advanced
AGA stages may have subclinical atherosclerosis more commonly compared to the
patients with beginning stages of AGA.

http://dergiler.ankara.edu.tr/dergiler/36/2055/21333.pdf

[NeedHairASAP]

Benign prostatic hyperplasia, metabolic syndrome and androgenic alopecia: Is there a

Benign prostatic hyperplasia, metabolic syndrome and androgenic alopecia: Is there a possible relationship?



Abstract
Objective

To evaluate the incidence of benign prostatic hyperplasia (BPH) and metabolic syndrome in patients with androgenetic alopecia (AGA) in comparison with those with no AGA, as several previous studies have reported inconsistent results of an association between metabolic syndrome and BPH with AGA.

Patients, subjects and methods

This cross-sectional study included 400 participants, divided into 300 patients diagnosed with AGA, with different grades according to Norwood–Hamilton classification, and 100 control subjects with no AGA. Criteria for diagnosis of metabolic syndrome according to Adult Treatment Panel-III criteria (waist circumference, blood pressure, fasting blood sugar, high-density lipoprotein and triglycerides), as well as criteria for diagnosis of BPH (prostatic volume, urine flow, and prostate-specific antigen) were assessed in all patients and compared with the control subjects.

Results

There were significant differences between the AGA and no-AGA groups for the following variables: waist circumference, body mass index, fibrinogen level, fasting blood sugar, cholesterol, C-reactive protein, erythrocyte sedimentation rate, and glycosylated haemoglobin. There was a significant difference in number of patients with AGA manifesting criteria of metabolic syndrome (51% vs 28%), as well as BPH diagnostic criteria (36% vs 6.8%) compared with the control subjects. Both BPH and metabolic syndrome were shown to be significant independent variables associated with AGA.

Conclusions

Dermatologists, urologists, and primary care physicians should monitor patients with early onset AGA for the development of urinary symptoms, to permit an earlier diagnosis of BPH; and for metabolic syndrome symptoms, to permit early diagnosis of cardiovascular risk factors.

http://www.sciencedirect.com/science/article/pii/S2090598X1600005X

[burtandernie]

OP says those who are genetically prone to getting hair loss. Not everyone who masturbates.

Of course he has to say that otherwise he would be factually wrong. At least now there is no major evidence proving him wrong, but nothing proving him right either.
IMO it can increase hair loss because it probably does increase T levels for a short time before and maybe after. That being said is it enough to make any difference? Probably not, and its not worth going over a theory with no evidence when so many other better explanations exist

[PatientlyWaiting]

This is such bro science BS it's not even funny. EVERYBODY after the age of 12 or 13 masturbates. Unless someone is going into the priesthood, literally everyone does it with regularity.

When you get older, you start having sex. That must be just as bad for hair then right? I mean it's setting off the same chain of biological responses. But no, for some reason everyone thinks it's just JO'ing that leads to hair loss. Show me a normal functioning person who doesn't do this and ill show you a liar. This thread shouldn't be anywhere near the cutting edge section, it's like something the Native Americans would have believed.

OP says those who are genetically prone to getting hair loss. Not everyone who masturbates.

[Trouse5858]

This is such bro science BS it's not even funny. EVERYBODY after the age of 12 or 13 masturbates. Unless someone is going into the priesthood, literally everyone does it with regularity.

When you get older, you start having sex. That must be just as bad for hair then right? I mean it's setting off the same chain of biological responses. But no, for some reason everyone thinks it's just JO'ing that leads to hair loss. Show me a normal functioning person who doesn't do this and ill show you a liar. This thread shouldn't be anywhere near the cutting edge section, it's like something the Native Americans would have believed.

[PatientlyWaiting]

I should be an NW10 then.

[Vic]

There are theories that suggest baldness is a defense mechanisms for other diseases: http://www.ncbi.nlm.nih.gov/pubmed/17910907

Androgenic alopecia may have evolved to protect men from prostate cancer by increasing skin exposure to ultraviolet radiation.



So there can be a link between vitamin D synthesis and DHT or other markers/hormones etc

Pair that with the fact that vitamin D synthesis needs cholesterol/fat to metabolize in skin, and there's an explanation for greasy scalp and hair loss. And consider that people are advised to wash hair frequently in case of hair loss which might worsen the symptoms since it strips the fats needed to make vitamin D.

The scalp is very vascularised which might explain the horseshoe pattern. http://www.erexam.org/wp-content/upl...asculature.png

Pair that with the fact that western society rarely gets any Sun, and vit D synthesis happens under UVB only (which is a narror timeframe, usually noon and in warm months when Sun is perpendicular to Earth - basically, the more inclined the angle it enters the atmosphere, the less UVB gets through), and that glass filters out UVB (so if you sit indoors behind a glass and Sun shines on you at noon, there will be 0 vitamin D synthesis).

I've read 90% of hair loss sufferers have a vitamin D deficiency. Getting 30 mins of sun is a part of my treatment method. Something I did notice with 30 min sun baths, when I applied minox before my sun time, my peach fuzz seemed darker. I've recently started using Emu oil before my sun time and I believe I'm seeing the beginning of some magic taking place. I can't say for sure its the combo of emu oil and sun which would be in line with what you wrote because I am now 7 months past my 3 month derma rolling treatment and it's basically the time supposed to start seeing results. Also have been using minox for 10 months. Whatever it is, this past month I've had an increase in hair at my temples. More and more terminal hairs popping out, darker and longer hairs that aren't terminal but way darker and stronger than peach fuzz.

[Tobias]

Sorry for digging it out, but too interesting=)

So, about the whole "is the rate of balding associated with bad health or even bad genes in general"...

If the studies that showed a correlation between balding rate and health are of good quality (which I didn´t check, but lets assume it is more than just a biased result), the this can mean two things: 1. Balding rate is a sign of bad genes. 2. Your overall health and lifestyle do have a strong effect on the rate of balding.

Number two is more likely and actually fantastic news and a very good reason for everyone to work on a less stressful and healthier lifestyle (don´t be fat, sleep enough, do some cardio, don´t worry too much about hair). Number one might be true for things that are directly caused by the setup of the androgen receptor gene (prostate cancer).

[garlicandginger]

If you all want to think that baldness is the only thing on planet earth that has nothing to do with evolution, than go for it. Baldness is in a class of it's own, and has no evolutionary significance what so ever.

It's purely a cosmetic thing that just so happens to correlate with really shitty genetic diseases, weak cells (going secenent prematurely) and also coincidentally manifests in a visual appearance that makes it harder for your to reproduce when compared with non-balding (and healthier) people.

There are theories that suggest baldness is a defense mechanisms for other diseases: http://www.ncbi.nlm.nih.gov/pubmed/17910907

Androgenic alopecia may have evolved to protect men from prostate cancer by increasing skin exposure to ultraviolet radiation.

Androgenic alopecia affects populations adapted to colder climate, and individuals at an age and hormonal status susceptible to prostate cancer. Male pattern baldness enhances absorption of UV radiation on the top of the head, an area directly exposed to sunlight during everyday activities. Ultraviolet radiation is reported to reduce the risk of advanced prostate cancer. Here I propose that progression of androgenic alopecia rather than being a risk factor is a finely tuned mechanism evolved to protect against prostate cancer.

So there can be a link between vitamin D synthesis and DHT or other markers/hormones etc

Pair that with the fact that vitamin D synthesis needs cholesterol/fat to metabolize in skin, and there's an explanation for greasy scalp and hair loss. And consider that people are advised to wash hair frequently in case of hair loss which might worsen the symptoms since it strips the fats needed to make vitamin D.

The scalp is very vascularised which might explain the horseshoe pattern. http://www.erexam.org/wp-content/upl...asculature.png

Pair that with the fact that western society rarely gets any Sun, and vit D synthesis happens under UVB only (which is a narror timeframe, usually noon and in warm months when Sun is perpendicular to Earth - basically, the more inclined the angle it enters the atmosphere, the less UVB gets through), and that glass filters out UVB (so if you sit indoors behind a glass and Sun shines on you at noon, there will be 0 vitamin D synthesis).

[d0072]

Appreciate the time you took to post this. Although I've read articles that say that masturbation has no effect on the severity of hairloss or even causing hairloss itself, I do suspect that there is in fact a connection. In my personal experience, I've been masturbating since about 12. That was around the y2k era right before online porn became pretty mainstream. I feel that in my case, masturbation has perhaps increased the amount of my hairloss. Deep down my intuition tells me that the whole reproductive system (Testosterone, semen production, prostate, etc.) has some correlation with hairloss. Maybe my guess is completely off, but this post certainly gives me some reassurance about not being completely wrong.

[Mickael]

Actually there is also a study on how identical twins that people thought were identical actually have different number of copies of some genes so they arent really identical. So a lot of studies using identical twins could be under false assumption that their genes are all identical.
All I know is castrated men never go bald. When someone proves to me MPB can be completely stopped or prevented through environment then I will follow that course, but no one has yet. There is no more convincing a case than proof MPB can be stopped through androgens by castration as hamilton proved decades ago. That to me proves its almost entirely androgen driven because the envirnment would vary for any castrated man yet hair loss never happens. Clearly its not environment as the major factor its something that changes naturally as you get older. Androgens fit that exactly and dut/propecia also prove that. Why randomly guess its the air, or stress, or a million other things that have no proof yet.
Maybe those things do contribute, but again no proof yet.

Yes it's obvious that androgen are the main cause of MPB. However, environment play a significant role on your androgen/hormonal balance and can influence how fast you go bald.

To make it quick:
Study A - It's been observed that men with early onset of baldness suffers from insulin resistance (pre-diabetics) and have low level of SHBG
Multiple Studies - It's been observed that the higher your insulin level is, the lower your level SHBG is, this likely means that high insulin level cause low SHBG level
Multiple Studies - Low SHBG level boost bio-availability of androgens including DHT.
Study B - AGA is linked to insulin resistance

This means that: Bad Lifestyle -> High Insulin -> Low SHBG -> More bioavailable DHT -> accelerated/aggressive hair loss

Even studies in twins with MPB have show that the environmental factors with high correlation of accelerating hair loss are all related to either hormones or insulin resistance.

It's been shown that people suffering from aggressive hair loss have more chance to get prostate cancer, diabetes, heart diseases and it's all related to insulin resistance.

[burtandernie]

Actually there is also a study on how identical twins that people thought were identical actually have different number of copies of some genes so they arent really identical. So a lot of studies using identical twins could be under false assumption that their genes are all identical.
All I know is castrated men never go bald. When someone proves to me MPB can be completely stopped or prevented through environment then I will follow that course, but no one has yet. There is no more convincing a case than proof MPB can be stopped through androgens by castration as hamilton proved decades ago. That to me proves its almost entirely androgen driven because the envirnment would vary for any castrated man yet hair loss never happens. Clearly its not environment as the major factor its something that changes naturally as you get older. Androgens fit that exactly and dut/propecia also prove that. Why randomly guess its the air, or stress, or a million other things that have no proof yet.
Maybe those things do contribute, but again no proof yet.

[Vic]

There are some studies involving identical twins with hair loss and they show that even with the same genes, people go bald at different rates. This means that you can significantly slow down hair loss if you put yourself within in a certain environment. From what I have seen, insulin resistance is a common factor, but there are other factors such as stress. At least, they are certainly a big factor in aggressive hair loss and early onset of baldness.

Well, you might not completely stop hair loss, but you can probably significantly slow down the rate at which you lose hairs based on your environment. This might has zero value, because I am a random person from the internet, but I myself suffered from an aggressive and early onset of baldness and I managed to significantly slow down my hair loss by fighting insulin resistance and other health issues related to modern lifestyle.

From my personal experience stress plays a HUGE roll. I had pretty much a full head of hair except for a muturing hairline. My fiancé turned out to be a slut, lost everything I ever worked for when the economy crashed(still had my hair) but soon after someone very close to me passed away and BOOM, 3-4 months later I lost 60-70% of my density. The only hair loss in my family was my cousin that had alopecia areata which grew back. Moms side don't even have maturing hairlines, full heads of hair/full density. Dads side has mature hairlines but full density.
Stress distroyed my hair!