Finasteride 0.25 mg twice per week. Works for me.
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Hopefully the photos I have posted showing my progress over the passed 7 months demonstrates that my modest dose of 0.5mg twice a week works (at least in the crown area, can't see much effect at the front unfortunately).
I'm not interested in making fun of people here as the prospect of baldness has been one of the most distressing things i have faced in my life so far. So chill out Kojak, we're all brothers here.Comment
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Then go to a specialist. They will list you in medical literature as a genetic freak.Hopefully the photos I have posted showing my progress over the passed 7 months demonstrates that my modest dose of 0.5mg twice a week works (at least in the crown area, can't see much effect at the front unfortunately).
I'm not interested in making fun of people here as the prospect of baldness has been one of the most distressing things i have faced in my life so far. So chill out Kojak, we're all brothers here.
I don't believe that 0.5 mg twice weekly could do anything significant. Your posts indicate that you seriously delusional.
You should post your DHT levels, by the way.Comment
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How do you explain the visibly shrunken crown i have achieved in the past 7 months? surely that warrants some merit as proof this dose has made a difference.
2 other members have posted in agreement that there is visible progress.
You should be happy with this result and adopt my dose Warlord. 1mg of Fin a week instead of 7mg will save you a heap of cash. You're welcome.Comment
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I see a head full of hair and no visible signs of hairloss.Comment
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Are you sure it isnt 5mg twice a week? I am taking 5mg pills and chop them into 4 and take a piece daily. This would mean you taking almost exactly what I am.Hopefully the photos I have posted showing my progress over the passed 7 months demonstrates that my modest dose of 0.5mg twice a week works (at least in the crown area, can't see much effect at the front unfortunately).
I'm not interested in making fun of people here as the prospect of baldness has been one of the most distressing things i have faced in my life so far. So chill out Kojak, we're all brothers here.
.5mg seems pretty darn low to me. Perhaps you misread it.Comment
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It sounds like you take Proscar.
I take Propecia which is a 1mg pill. I bought a month's supply (28 pills) and funnily enough I am still on my first pack....although I'm now down to my last pill. I'm about ready to buy my second ever packet of Propecia (which in Australia is about 100 bucks a pack).Comment
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The excess in testosterone is expected, your HTPA system is compensating for the reduction in DHT with extra testosterone.
I dont want to scare you, but this string of drug induced messages associated with the HTPA system is often what sets off the endocrinal 'crash' when coming off the drug, your body has nothing to do with the extra testosterone but to convert it into estrogen which usually results in the feminising effects of the drug. Now if you were to take an estrogen blocker you'd just end up with high endogenous levels of testosterone which could damage your liver - the fact that your testosterone levels are consistently high due to the effects of this drug is what sometimes causes a total shutdown of endogenous testosterone production when coming off the drug, setting you up for a lifetime of hypogonadism.
I also wouldn't trust the 2% side effects statistic being that Merck also stated that "DHT does not have much use in the male body" - I have been rolf'ing at that for a few years now.
DHT is one of the most important male hormones, the very fact that it determined your gender when you were in a foetal state should indicate that.
Good luck.Comment
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Fearmongering! The increase of testosterone induced by 5-AR is small, much smaller than e. g. in anti-estrogens that are capable to elevate your testosterone levels by more than 100%. Hence the risk of gynecomastia is small as well, about 1.3%.The excess in testosterone is expected, your HTPA system is compensating for the reduction in DHT with extra testosterone.
I dont want to scare you, but this string of drug induced messages associated with the HTPA system is often what sets off the endocrinal 'crash' when coming off the drug, your body has nothing to do with the extra testosterone but to convert it into estrogen which usually results in the feminising effects of the drug. Now if you were to take an estrogen blocker you'd just end up with high endogenous levels of testosterone which could damage your liver - the fact that your testosterone levels are consistently high due to the effects of this drug is what sometimes causes a total shutdown of endogenous testosterone production when coming off the drug, setting you up for a lifetime of hypogonadism.
I also wouldn't trust the 2% side effects statistic being that Merck also stated that "DHT does not have much use in the male body" - I have been rolf'ing at that for a few years now.
DHT is one of the most important male hormones, the very fact that it determined your gender when you were in a foetal state should indicate that.
Good luck.
And as for DHT, it has no important function after adolescence - if you don't consider baldness, prostate enlargement and prostate cancer as things without which you can't be alive.Comment

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