Iv read so many good reviews on this around web, ancient people used oils on hair so this look promising I'm going to give it a go.
RU is working wonders on Hairloss Help
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Those pictures I posted of hairline, the before picture was taken while I had done great on 7 months regaine foam, the after I added finesteride and 12.5% cream my hairloss decreased and my started getting thicker all my scalp issues like tingling, itchy etc went away. Also bare in mind some people who suffer MPB get different effects, some people getting itchy sclp, tingly scalp where as others just experience hairloss and no inflammation.Leave a comment:
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I was too afraid to take pictures as it made me more depressed, I'm 100% MPB gene.
Here's some pictures,
This is actually alopecia areata
This Is MPB, this was before any meds and things started to look worse, the picture does no justice but my partner knows exactly how thin it was.
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I'm asian, I was balding thanks to propecia, it sucks because my brother and dad both have really great thick hair but I have my bloody mums genes. One of my mums brothers are bald, if I stop propecia I get sevre hairloss, scalp tingles bad all over, itches etc I'm just lucky I caught it on time, but I need something to hold it till I die otherwise my last option would be a transplant. I know you guys think it looks great but unfortunatly it's down to meds not genes
. Oh and I was receding to a v shape I don't have pictures but those pictures were after minoxidol and finesteride prior I was losing ground hard.
There is a difference between balding and shedding, you dont have mpb from the looks of it, neither do 50% of us who are on here! FACT.Leave a comment:
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I'm asian, I was balding thanks to propecia, it sucks because my brother and dad both have really great thick hair but I have my bloody mums genes. One of my mums brothers are bald, if I stop propecia I get sevre hairloss, scalp tingles bad all over, itches etc I'm just lucky I caught it on time, but I need something to hold it till I die otherwise my last option would be a transplant. I know you guys think it looks great but unfortunatly it's down to meds not genes
. Oh and I was receding to a v shape I don't have pictures but those pictures were after minoxidol and finesteride prior I was losing ground hard.
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Jazz, what nationality are you? With your thick hair and NW0, don't see why you're even here.Leave a comment:
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He's probably jsut upset and looking at the 0.01% side effects on one guy as a bad for everyone else, think about finesteride millions users and 2% get sides etc. Minoxidol was same I had funny chest pains now they gone along time ago, I think it's always best to use very small dose first see how ones body reacts etc as you need to let any drug get use to your body otherwise a high dose can be a shock and cause side effects.
This is why people who take minox have these "chest pains". It's just a homeostatic reflex that occurs in our body. The opposite is also true if we were to vasoconstrict our vessels, hr would decrease.Leave a comment:
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I'm going try this as I use hair oils ever now and then and they work, ancient people used oils aswell. But this oil is special iv read so many good stuff on it, all oils around world are mixed in one.
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Talking out of your @$$ again I see. Actually, their mechanisms aren't really that much different. Finasteride is a competitive inhibitor of DHT, whereas RU is a NON-competitive inhibitor of DHT.
Do you know what Pheochromocytoma is? You probably don't...but the drug of choice for treating this condition is Phenoxybenzamine, and not Phentolamine. Why? PBenz is a NON-competitive inhibitor, whereas Ptol is a competitive inhibitor. Having a NON-CI means the drug will absolutely prevent DHT from binding to the receptors, regardless of how much substrate that develops. With competitive inhibitors, physiological increases of DHT simply knock Finasteride off from the binding sites of it's receptors, and DHT can resume binding to the location allowing further miniaturization and loss of hair follicles.Leave a comment:
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, here's a picture of my hairline before and after using 12.5% cream. Please bare in mind I lost some hairs which are slowly coming back from a shed. I read good stuff about Dr Klein so lets hope it works when I get my bottles, he did say it should be posted next week, I'm stopping the 12.5% as here in the uk as that belgravia center charge way too much money, and for my DR KLEIN does great deals and prices.
Before 12.5%
After
Now;
I have loads pictures il post a thread one day I just want to try everything to get my regime right before I start posting more pictures lol.
Also I get alopecia areata, since fitting a reverse osmosis here in the uk and water system etc my alopecia patches are growing back it was bigger then a 2p coin, my hairdresser was right so many people have alopecia areata it's hard water full of chemicals we tested it. Now I'm drinking clean water and showering I don't get no more muscle cramps in the gym and the hairs growing back here,
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Well then what about higher dose topical minox at 15% it could if used twice a day or less cause side effects thats why fda took it off the market as we know 5 is the max fda and all over the world and even that can cause sides.
I did use minox 4 and 5 with progestorone added and many clinics use this and i have never had any problems.Leave a comment:
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I am not talking about finasteride. I am talking mostly about RU being life-threatening. I would advise everyone to abstain from it or be very careful with such drug.
It appears the mechanisms of finasteride and RU are different. I would say finasteride is much safer and that's why it made it to market but it doesn't mean it's completely safe.
[As per Dr. Gho's written request, this comment has been removed.]
Do you know what Pheochromocytoma is? You probably don't...but the drug of choice for treating this condition is Phenoxybenzamine, and not Phentolamine. Why? PBenz is a NON-competitive inhibitor, whereas Ptol is a competitive inhibitor. Having a NON-CI means the drug will absolutely prevent DHT from binding to the receptors, regardless of how much substrate that develops. With competitive inhibitors, physiological increases of DHT simply knock Finasteride off from the binding sites of it's receptors, and DHT can resume binding to the location allowing further miniaturization and loss of hair follicles.Leave a comment:
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