Hair loss Is preventable and reversible. A must Read!

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  • beatinghairloss
    replied
    Originally posted by 2020
    I give up

    Your theory is right: skull expansion triggered by DHT that thickens the galea and restricts bloodflow and something something shrinks follicles. You're exactly right. Good luck with whatever massages and wands you're planing to use. I'm out
    Hahah what? I never said DHT causes skull expansion. Stop messing around we have work to do. No really though what do you think about this site. I just found it? Do you think this guys full of crap?

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  • 2020
    replied
    I give up

    Your theory is right: skull expansion triggered by DHT that thickens the galea and restricts bloodflow and something something shrinks follicles. You're exactly right. Good luck with whatever massages and wands you're planing to use. I'm out

    Leave a comment:


  • beatinghairloss
    replied
    Do you have links to the actaul studies, pictues, where or how they removed the galea? Where did this relieve pressure? Was pressure even measured? Did any of the people have any reasonable scalp fat increase? What where the follow up protocols? Agains balding is a multi approach I would need much more then your word for it. I am not saying I can not be convinced I just have not encountered one single reasonable study.

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  • beatinghairloss
    replied
    Originally posted by 2020
    About which one of my posts are you talking about? Hair follicles that aren't in anagen phase need less blood flow - that's how follicles work and that's just a fact.


    What's wrong with my previous study? Removal of galea was performed on 56 people. MPB was not stopped. What do you make of that?
    I don't see how removing galea would help this to me would add to the problem you want to increase the fat in the scalp by adding. Blood flow in the study I showed measures capillary blood flow not follicle blood flow that would be impossible so while your statement is true it wouldn't effect blood to the scalp.

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  • 2020
    replied
    Originally posted by beatinghairloss
    Again small studies no results mostly assumptions I need more
    About which one of my posts are you talking about? Hair follicles that aren't in anagen phase need less blood flow - that's how follicles work and that's just a fact.


    What's wrong with my previous study? Removal of galea was performed on 56 people. MPB was not stopped. What do you make of that?

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  • beatinghairloss
    replied
    Originally posted by 2020
    btw, you do realize that hairs anagen(growth) phase actually stimulate the blood supply??? Once your hair is in catagen phase, blood supply is naturally lower...



    this explains why bald people with less HAIR IN ANAGEN PHASE will have less bloodflow in that area....


    are we done now???
    Again small studies no results mostly assumptions I need more

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  • 2020
    replied
    btw, you do realize that hairs anagen(growth) phase actually stimulate the blood supply??? Once your hair is in catagen phase, blood supply is naturally lower...

    Catagen phase
    Signals sent out by the body determine when the anagen phase ends and the catagen phase begins. The catagen phase, also known as the transitional phase, allows the follicle to, in a sense, renew itself. During this time, which lasts about two weeks, the hair follicle shrinks due to disintegration and the papilla detaches and "rests", cutting the hair strand off from its nourishing blood supply.
    this explains why bald people with less HAIR IN ANAGEN PHASE will have less bloodflow in that area....


    are we done now???

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  • 2020
    replied
    During the past few decades, several hypotheses concerning the etiology of male pattern baldness (MPB) have been presented. In 1933, Wadel reported findings of decreased motility of the scalp. He was convinced that this decrease was due to the fact that in MPB patients the scalp is both frontally and sagittally too short, and thus it has to be stretched like a too-small cap to cover the relatively too-big skull. For hair nutrition and rooting this persisting tension creates unbearable conditions, leading to gradual loss of hair. In 1935 he wrote that MPB is the end result of the tension atrophy of the scalp covering the galea aponeurotica. This atrophy is caused by a disproportion between the skull bone and the galea aponeurotica, due to an isolated growth of the skull bone to which the tendon-like structure of the galea is not able to adapt. He reported excellent results in the treatment of MPB with 'loosening' massage to the scalp.

    "In 1941, Kessler started experimental work with frontal galeotomies in order to reduce the supposed increased tension of the galea aponeurotica. In 1961 he reported a success rate of 87% with this treatment of MPB. At that time this operation was popular in Europe. In 1963, Ponten reported that after frontal galeotomy he could not find any objective improvement in his 56 patients and he still holds this view concerning this operation (personal communication, 1976).

    "The present author has seen several patients who have undergone frontal galeotomy and later developed an advanced degree of MPB. The popularity of this operation has waned.
    "

    "In the receding hairline and in the graft taken from it the loss of hairs remains synchronous even though the latter is transplanted to a remote skin area. In MPB the 'balding clock' in the follicle or in its very close surrounding keeps time even when the follicle is transplanted to the skin of the forearm. The presence or absence of the galea aponeurotica does not influence the balding process in MPB. Nor does the supposed increased tension of the scalp or its muscles or a diminished vascular supply to the scalp have an effect on balding. Neither do any other factors localized to the head cause balding. The cause seems to lie in the follicle itself or its very close surrounding. The graft taken from the denuded area did not grow new hairs, and so the MPB process of the hair follicle is not reversed by a change in its location on the human body.
    are we done now?

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  • 2020
    replied
    Originally posted by beatinghairloss
    My friend it is not about thinking as much as it is assuming. You assume hair loss is hair per hair genetic predisposition but if this where true variation would cause this hair loss to be scatterd to the entire body.
    follicles on your body are not programmed in such way.

    Originally posted by beatinghairloss
    Even if it was scatterd across the entire head it would be more believable.
    it's called diffuse thinning

    Originally posted by beatinghairloss
    Unfortunately you assume this fact based on one test one person this is terrible scientific judgment. You should demand more.
    so they just got extremely lucky with that person huh? What makes you think they couldn't repeat that experiment with the same results on a thousand people?



    Originally posted by beatinghairloss
    The area men experience balding has localized similarities that are way past mere coincidence. The galea is the key but more importantly it's blood flow via unintentional looseness and movement of the scalp. Genetic is merely the shape of your head and muscles in the head
    BUT HAIR TRANSPLANTS WORK! I found you a patient whose hair persisted on a MPB area with "poor circulation" for 30 years!!! Explain that

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  • beatinghairloss
    replied
    Originally posted by 2020
    what makes you think such experiment won't work on 1000 people?
    My friend it is not about thinking as much as it is assuming. You assume hair loss is hair per hair genetic predisposition but if this where true variation would cause this hair loss to be scatterd to the entire body. Even if it was scatterd across the entire head it would be more believable. Unfortunately you assume this fact based on one test one person this is terrible scientific judgment. You should demand more. The area men experience balding has localized similarities that are way past mere coincidence. The galea is the key but more importantly it's blood flow via unintentional looseness and movement of the scalp. Genetic is merely the shape of your head and muscles in the head

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  • 2020
    replied
    Originally posted by beatinghairloss
    what makes you think such experiment won't work on 1000 people?

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  • 25 going on 65
    replied
    This thread is just a bad joke at this point.

    Occam's Razor. Use it.

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  • beatinghairloss
    replied
    Originally posted by 2020
    I already gave you one:

    two follicles were taken out of a subject - one from donor area, and one from MPB area.

    Both of those follicles were implanted on subjects arm.
    Follicle from MPB area shrunk, while follicle from donor area grew just fine.

    ^ doesn't that prove that LOCATION doesn't matter and the problem is actually inside individual follicles that tells them to shrink???

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  • Davey Jones
    replied
    I'm gonna assume the bacteria they're talking about is candida. The autoimmune response is associated with (or more accurately, it is) chronic, systemic inflammation. Chronic inflammation is bad, and I go pretty far out of my way to counter that, including measures to restrict the overdevelopment of candida.

    I'm pretty unconvinced that inflammation has much to do with balding. Or, even if it is, that we can restrict it enough to have an effect on balding. I'd like to think that if we could, I would have noticed something by now. Buuuuut, maybe not.

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  • 2020
    replied
    what is this?

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